Creation of New Brain Cells May Be Limited, Mouse Study Shows
Many researchers have long assumed that most stem cells in the body can produce new cells indefinitely, but new research at UCSF shows that this is not the case in the brain.
![](/sites/default/files/styles/news_card__image/public/fields/field_insert_file/news/Obernier_Mouse_Neurons_0.jpg)
University of California San Francisco
Give to UCSFMany researchers have long assumed that most stem cells in the body can produce new cells indefinitely, but new research at UCSF shows that this is not the case in the brain.
An international team of researchers has shown that two different compounds, can safely and effectively be added to treatment regimens to block transmission of the most common form of malaria in Africa.
A UCSF research team has found that while banning flame-retardant chemicals initially led to a reduction in exposure, a disturbing trend is emerging of exposure leveling off or even rising again.
UCSF researchers have identified the buildup of one brain chemical as a key culprit behind age-related learning and memory impairments. Tuning levels of this chemical in the worm C. elegans, they could delay and even reverse the declines of old age.
A new study shows that an immune signal named interleukin 33 plays a crucial role in allowing the brain to maintain the optimal number of synapses during the development of the central nervous system.
For the first time, neuroscientists have identified “anxiety” cells deep inside the brain.
Stretches of DNA that make us uniquely human are partly responsible for controlling neuron growth, according to new research from the Gladstone Institutes and UCSF.
Denal Dubal, an associate professor of neurology at UCSF, thinks we can use the science of aging to help stave off these neurodegenerative diseases.
UCSF scientists have invented a technique that lets them precisely and reversibly disrupt the action of specific cellular proteins at a microscopic scale by making them split apart when illuminated with blue light.
Type 2 diabetes is known to be a risk factor for bone fractures – but exactly how diabetes makes bones more fragile has been unclear.
Researchers at UCSF have found a way to attack one of the most common drivers of lung, colorectal, and pancreatic cancer by targeting the proteins it produces on the outside of the cell.
A scientifically based approach that includes a tooth-decay risk assessment, aggressive preventive measures and conservative restorations can dramatically reduce decay in community dental practices.
UCSF researchers have discovered a new biological pathway in fat cells that could explain why some people with obesity are at high risk for metabolic diseases such as type 2 diabetes.
SF CAN is targeting the five most common cancers which collectively account for half of all new cancers in San Francisco.
More and more, the promise of EHRs transforming data into knowledge is beginning to bear fruit.