UCSF Researcher Identifies Risk Genes for ALS
The largest analysis to date of genetic data in ALS has identified two previously unrecognized genetic risks that are significantly associated with the disease.
University of California San Francisco
Give to UCSFThe largest analysis to date of genetic data in ALS has identified two previously unrecognized genetic risks that are significantly associated with the disease.
UCSF researchers have shown that an experimental brain boosting drug, ISRIB, acts like a molecular staple, pinning together parts of a much larger protein involved in cellular stress.
The 2018 Grad Slam competition challenged PhD students to use straightforward yet engaging language that non-specialists can understand to describe their intricate research – in three minutes or less.
UCSF scientists uncovered a common genetic driver of aggressive meningiomas, which could help clinicians detect dangerous cancers earlier and lead to new therapies.
UCSF’s School of Medicine placed in the top five nationally in this year’s U.S. News & World Report survey of best graduate and professional schools. UCSF’s biomedical science PhD programs were among the top 10, and the School of Nursing was also highly ranked.
A new study in mice reveals how a gene mutation seen in human short-sleepers may allow them to survive and thrive on just a few hours of sleep.
A study of patients with early Parkinson’s disease found that groups with lower levels of vitamin B12 faced on average a more rapid acceleration of both motor and cognitive symptoms.
UCSF scientists have shown that in the human hippocampus neurogenesis declines throughout childhood and is undetectable in adults.
UCSF received more than $593.9 million in federal funding from the National Institutes of Health in 2017 for research across multiple health-science arenas at the University.
New research led by David Solomon, an assistant professor in the Department of Pathology at UCSF, provides much-needed targeted treatment options for patients whose tumors cannot be surgically removed.
Loss of an enzyme that modifies gene activity to promote brain regeneration may be partly responsible for age-related cognitive decline, according to new research in laboratory mice by UCSF.
Many researchers have long assumed that most stem cells in the body can produce new cells indefinitely, but new research at UCSF shows that this is not the case in the brain.
UCSF researchers have identified the buildup of one brain chemical as a key culprit behind age-related learning and memory impairments. Tuning levels of this chemical in the worm C. elegans, they could delay and even reverse the declines of old age.
A new study shows that an immune signal named interleukin 33 plays a crucial role in allowing the brain to maintain the optimal number of synapses during the development of the central nervous system.
For the first time, neuroscientists have identified “anxiety” cells deep inside the brain.