Latest News

November 07, 2013
When it comes to genes, a UCSF scientist wants to turn conventional wisdom about human and bacterial evolution on its head.
October 11, 2013
The way cells divide to form new cells – to support growth, to repair damaged tissues, or simply to maintain our healthy adult functioning – is controlled in previously unsuspected ways, UCSF researchers have discovered.
September 15, 2013
A new link between meal times and daily changes in the immune system has been identified by UCSF researchers, and has led them to question assumptions about the roles of specific immune cells in infection and allergy.
August 19, 2013
UCSF scientists working in the lab used a chemical found in an anti-wrinkle cream to prevent the death of nerve cells damaged by mutations that cause an inherited form of Parkinson’s disease.
August 12, 2013
Researchers have probed deep into the cell’s genome to begin learning the “grammar” that helps determine whether or not a gene gets switched on to make the protein it encodes, advancing efforts to use gene and cell-based therapies to treat disease.
August 08, 2013
A team of investigators led by UCSF and the Gladstone Institutes has found a way to map an enzyme’s underlying molecular machinery, revealing patterns that could allow them to predict how an enzyme behaves – and what happens when this process disrupted.
July 03, 2013
The ability to form blood vessels is one of evolution’s crowning achievements. Now, scientists at the Gladstone Institutes have identified the molecular signals that direct the process of committing endothelial cells to become arteries or veins during embryonic development.
June 27, 2013
A new UCSF study highlights the potential importance of the vast majority of human DNA that lies outside of genes within the cell.
June 17, 2013
Memory improved in mice injected with a small, drug-like molecule discovered by UCSF researchers studying how cells respond to biological stress.
April 18, 2013
UCSF scientists have discovered that muscle repair requires the action of two types of cells better known for causing inflammation and forming fat.

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